呼吸系统影像学Imaging of respiratory systemWord文档格式.docx
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呼吸系统影像学Imaging of respiratory systemWord文档格式.docx
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thelungtissuedensityincreases,thevolumeshrinks,thetriangleistriangular,theedgeisclearandsharp,thetippointstothelungdoor,thebaseisattachedtotheseptumorthechestwall,enhancedafterenhancement.Theadjacentstructureiscompensatedfor.2.EmphysemaandlunghyperinflationPrinciple:
thebronchiolesarenotcompletelyobstructed,andthelivingflapisventilated.Thetwolungsarewidelydistributedwiththeirreversibledamageofthealveolarwall.Thelatterisnotassociatedwiththedestructionofthealveolarwallandisoftenoverinflatedwithonesideoronelobe.ThepathologicalanatomycanbedividedintosmallleafcentralemphysemaandpulmonaryemphysemaattheintervalofpulmonaryemphysemaThree,theconsolidationofthelung(referstotheterminalbronchiolesgasbeyondlacunainairwerereplacedbyaliquid,cellortissuepathological.Involvingtherangecanbeacini,lobular,pulmonarylobectomyorparagraph.)CTmanifestations:
glandularnodulesandflakymarginblurredimages.Theshapeofthelungsectionorthedistributionofthepulmonarylobe,whichcanbeseenintheinteriorofthebloodvessels,canbeseenasalarge,high-densitysolidshadow,whichcanbeseenasbronchimeteorology.Bronchimeteorology:
thesolidlungtissueissetoffbythebronchialbronchialfacies.Intherealchangearea,thetransparenttrachealshapeofthebranchisknownasbronchialmeteorology.Alsoknownasairbronchialsigns,includingbronchialsigns(commonin:
pneumonia(largeleaf),pulmonaryedema,pulmonarytuberculosis,pulmonarycontusion,pulmonaryhemorrhage,pulmonaryinfarction)4.Pulmonarymasses(2cmindiameter,withclear,rounded,roundedshapes.Seeninneoplasticlesions(benign,malignant)andnon-neoplasticlesions(nodules,inflammatorypseudotumor).Characteristicsofbenignlungtumors:
circularorelliptic,marginsmoothsharpseeexplosionincornfigureadiposetissuecalcificationormildenhancementCTvalueincreaseslessthan20hu,below3cmindiameter,masssurroundedbysatellitekitchenadjacentpleuralthickeningandadhesionMalignantlungtumorcharacteristics:
1.Marginallobesormarks2.Thereareradiated,shortandthinburrsaround3.Theadjacentpleuralmembraneisconcavetothemass4.Theinnerbloodvesselsofthemasses5.Thebronchialtubeofthetumoristruncatedornarrow,andthewallthickens6.Enlargementofthemediastinallymphnode,shorterthan1-1.5cm7.Theemptyinnerwallformedisirregularandhaswallnodules8.Thereare1-2mmvacuolesandairbronchogenicsignsinthemassThechestwall,thepleuraanddistantmetastasesVoidandcavityCavitation:
necroticliquefactionofdiseasedtissueinthelungisformedbytheremovalofbronchialtubes.Common:
pulmonaryabscess,tuberculosis,lungcancer,staphylococcalpneumonia,fungaldisease.Above3~10mmisthickwallhole,3mmbelowisthinwallhole.Cavity:
thepulmonarycavitywasenlargedwiththepathologicandthelocalgasswellingandlocalpneumothoraxcausedthecollectionofalveolarwalls.Common:
pulmonaryalveolar,branched,pneumatocyst,pulmonarycyst,etc.Cavitywallthicknessaboutlmm.Six,pulmonaryinterstitiallesions(mainlyreferstoinfringementofinterstitiallungdisease,infactisoftenaccompaniedbythechangeofthelungparenchyma.Atthesametimewhendiseaseandpulmonaryinterstitial,canbeproducedwithinthepulmonaryinterstitialgappathologicalliquid,inflammatoryinfiltration,granulationtissue,fibroustissueandtumortissue.Differentinfringementofpulmonaryinterstitialdiseaseareacanbefoundin:
chronicidiopathicinterstitialpneumonia,interstitialfibrosis,sarcoidosis,andlymphangiticspreadofcarcinoma,connectivetissuedisease,pneumoconiosis,etc.)CTmanifestations:
Interfacialfeatures:
theinterfacebetweenthickeningandaeropulmonarytissuecontrast.Thebronchus,thebloodvesselsarethickening.ThickeningoftheinterlobularseptumandcentrilobularstructureExtensionoftractionandtractionThechangesofthegrindingglass:
pathologicalcanbeasmallamountofseepageinthealveoli,theswellingofthealveolarwallortheinflammationofthealveolarseptum.
(1)interfacesign:
forearlyperformance.Inflatablelungandbronchus,bloodvessels,dirtyappearedirregular,notsmoothinterfacebetweenpleura,foundinmostofthepulmonaryinterstitiallesions(89%)andidiopathicpulmonaryfibrosis(98%).Nonspecific.2.Thickeningoflobularseptum:
oneofthecharacteristicsofpulmonaryinterstitiallesions.Itisshownastheverticallineshadowofthepleuralsurface,whichis1-2cminlengthandis0.5-2cmapart,andisalsoknownastheintervalline,whichisinthecentralareaofthelunginapolygonandarch.Oftenaccompaniedbylobulestructuredeformation,irregular,smallleafinternallinelikeshadow.3.Thickeningofperibronchovascularinterstitium:
acommonsignofinvasivelungdisease.Themostcommonlesionsareinterstitialpulmonaryedema,primaryandprogressivelymphaticlesionssuchaslymphangiopathy,sarcoidosisandpulmonaryfibrosisNodules(2-5mmnodules)4.Smallleafinternalline:
thethickeningofinterstitialthickeningofthesmallleafiscausedbythinmeshandscreenshape,whichiscommoninpulmonaryfibrosisSmallvesiclesandhoneycombs:
smallvesiclesarecircular,andhaveaclearboundarywall,commonlyseeninidiopathicpulmonaryfibrosis,fibroticalveoli,asbestos,andchronicallergicpneumonia.Cysticlumenisseenintheendoffibrosis,oftenaccompaniedbystructuraldeformationandtraction.6,nodularshadows:
1~10mmsmallnodularshadows,oftenhasawellasinterstitiallesions,boundaryisnotclearorthegroundglassdensitymainlyaffectedgasgap,buttoidentifythestromaorsubstantialinvolvementisoftendifficult7.Groundglassdensity:
themistdensityincreases,andthebronchovascularstructurecanbedistinguished.Canbecausedbyairgaplesions,interstitialthickeningorboth.Iftherearenootherfibrosissigns,itisoftensuggestedtobearecoverablelesion.Inchronicinvasivelunglesionsfoundinidiopathicpulmonaryfibrosis,withhairincollagenvasculardiseasefibrosisalveolarinflammation,desquamationofinterstitialpneumonia,hypersensitivitypneumonitis,sarcoidosis,alveolarproteinADcool-headeddisease.)Vii.PleurallesionsPleuraleffusionandfluidpneumothoraxThepleuralcavityisapotentialnegativepressurecavity,andthereisasmallamountofliquidinnormal,whichcanbeusedtolubricatethechestPleuraltumors:
pleuraltumorscanbefoundinthepleuraprimaryormetastatictumors,suchaspleuralmesothelioma(benignandmalignant),malignantthymomaextendsalongthepleuraandlungcancermetastasis,etc.,alsovisibleinthetumorlesionsuchasmachinesexpyothoraxandpleuralplaquesareasbestosisPneumothorax:
CTimage(lungwindow)isseeninthevolumeofthegas,whichcanbeseenindifferentareasofthelung,withnolungtexture,andtheinnermarginofthecompressedlungedgeBasicshadowdiagnosisofmajorcommondiseasesinthechestChronicbronchitisPneumonia(largeleaf)LungabscessTuberculosis(TB)bronchiectasisLungcancerMediastinaltumorCommoncardiovasculardisease1.ChronicbronchitisChroniccoughandphlegm-twoconsecutiveyears,threemonthsinayear--------------------------------------------Causes:
smoking,infection,airpollutionClinicaldiagnosisisthemain,imagingdiagnosisisauxiliaryThemainmanifestationsofCTare:
pulmonarytexturedisorder,distortion-webweaves,thickeningofbronchialwall-tracksign,secondaryemphysemaCTisusedtoidentifyotherdiseasesthatcausecoughandcough,suchasbronchiectasis,lungcancer,andpulmonaryinfection2,pneumonia
(1)largeleafpneumonia(mainlycausedbypneumococcus,alsoknownasklebsiella,legionellainfection;
typicallobulepneumoniainyoungpeople)CTmanifestations:
hyperemiaperiod:
itcanbefoundthatthelesionareaisintheshadowofthegroundglass,andtheedgesareblurred.Bloodvesselsinthediseasedareaarestillvisible.Theperiodofrealchange:
alarge,flakyshadowthatcanbeseeninthedistributionoflungsegmentsorlungleaves,visiblebronchialmeteorology.Dissipationperiod:
withtheabsorptionofthelesion,thedensityoftherealvariableshadowdecreases,andthespecularshadowisscatteredindifferentsizesThefinalabsorptioniscomplete
(2)lobule(bronchial)pneumoniaBronchialenlargementofbloodvesselbundleintheconventionalCTscanbothlungspart,visiblenodularshadowsandflakeofdifferentsize,about1~2cmsize,edgeblur,multiplesmallshapeshadowbetweendopedwithgaslungtissue.(3)interstitialpneumoniaEarlyormildcasesofinterstitialpneumonia,canbeperformedonbothsidesofthebronchialenlargementofbloodvesselbundle,accompaniedbyshadowgrindingofglasssamples,onbehalfofthebronchialaroundwithinterstitialinflammatoryinfiltratesandalveoliinflammatoryinfiltratesandasmallamountofeffusion.Thecontrastcanbeaccompaniedbyasmallleaf,whichcanbeseenasaspecularshadow.Thepulmonaryandmediastinallymphnodesmayincrease.Iii.Pulmonaryabscess(progressofpulmonaryinfection)Pulmonaryabscessisthelocalnecrosisandsuppurativeinflammationcausedbythesuppurativebacteria.Clin
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